I keep almost fainting?!
Question: I keep almost fainting!?
Like i was at the waterpark and we were in line for food and i started falling backwards and i couldnt see anything all i heard was "sam sam stay with me were almost to the table" and sometimes i get out of the shower i get really light headed and i have to sit down for like 15 minutes!. itll just happen at random times and i dont get it!. any idea why this is happening!?
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Answers:
Low blood pressure!.
Common factors are heat, first day of your period, getting up quickly, getting up from lying down, dehydration, and not eating recently enough!.
You may find it resolves almost immediately if you either lie down or when you sit, put your head between your knees!. This gets oxygenated blood to your brain the quickest!.Www@Answer-Health@Com
Common factors are heat, first day of your period, getting up quickly, getting up from lying down, dehydration, and not eating recently enough!.
You may find it resolves almost immediately if you either lie down or when you sit, put your head between your knees!. This gets oxygenated blood to your brain the quickest!.Www@Answer-Health@Com
Vasovagal reaction!. Go see your doctor!.!.!.!.!.!. enough said
Well done to the person that looked up Vasovagal reaction and placed everything you could find on here to make this young girl crap herself !.!.!.!. well done!.
Basically a vasovagal reaction is the most common form of fainting usually brought on from emotional stress, pain or dehydration and for standing for long periods of time!. Fainting is due to a drop in blood pressure which decreases blood flow to the brain and results in loss of concsiousness!. So all up its a common cause of fainting!. Most faining episodes are not life threatening!.Www@Answer-Health@Com
Well done to the person that looked up Vasovagal reaction and placed everything you could find on here to make this young girl crap herself !.!.!.!. well done!.
Basically a vasovagal reaction is the most common form of fainting usually brought on from emotional stress, pain or dehydration and for standing for long periods of time!. Fainting is due to a drop in blood pressure which decreases blood flow to the brain and results in loss of concsiousness!. So all up its a common cause of fainting!. Most faining episodes are not life threatening!.Www@Answer-Health@Com
i think you should go to the doctor and do a detailed clinical check , it is the right thing to do , because there are million of things that can cause fainting , so listen to me and go to the doctor , you will not lose anything!. Www@Answer-Health@Com
Obviously, if you keep fainting then you should see a doctor to find out the reason for it!. It could be due to lots of things, some of them quite serious!.Www@Answer-Health@Com
Dehydration, maybe low blood pressure!. Whatever it is, you should go see a doctor about it as soon as possible!. Www@Answer-Health@Com
It could be you need to drink a lot of water!. OR maybe be tested for diabeties!.Www@Answer-Health@Com
could be dehydrationWww@Answer-Health@Com
The condition you are describing is called vasovagal syncope (fainting)!. here is what I found on it from wikipedia!.
Vasovagal syncope is the most common type of fainting!. There are a number of different syncope syndromes which all fall under the umbrella of vasovagal syncope!. The common element among these conditions is the central mechanism leading to loss of consciousness!. The differences among them are in the factors which trigger this mechanism!.
Contents [hide]
1 Triggers
2 Features
3 Pathophysiology and mechanism
4 Diagnosis
5 Prognosis
6 Treatment
7 References
8 Other sources
9 External links
[edit]Triggers
Typical triggers for vasovagal syncope include:[1]
prolonged standing or upright sitting, particularly when standing with legs in a locked position for long periods of time!. Avoidance of long-term locking of one's legs in the standing position is taught in the military, as well as marching bands and drill teams!.
stress
any painful or unpleasant stimuli, such as
giving a blood donation or watching someone give one
watching someone experience pain
watching/experiencing medical procedures
sight of blood
hyperthermia, a prolonged exposure to heat
(even occasions of slight discomfort, such as dental and eye examinations)
sudden onset of extreme emotions
hunger
nausea or vomiting
dehydration
urination ('micturition syncope') or defecation ('defecation syncope')
swallowing ('swallowing syncope')
coughing ('cough syncope')
abdominal straining or 'bearing down' (as in defecation)
random onsets due to nerve malfunctions
Pressing upon certain places on the throat, sinuses, and eyes
High altitude
The use of certain drugs that affect blood pressure, such as amphetamine
after a period of intense laughter[2]
[edit]Features
People with vasovagal syncope typically have recurrent episodes, usually when exposed to a specific trigger!. The initial episode often occurs when the person is a teenager, then recurs in clusters throughout his or her life!. Prior to losing consciousness, the individual frequently experiences a prodrome of symptoms such as lightheadedness, nausea, sweating, ringing in the ears, uncomfortable feeling in the heart, weakness and visual disturbances!. These last for at least a few seconds before consciousness is lost, which typically happens when the person is sitting up or standing!. When they pass out, they fall down; and when in this position, effective blood flow to the brain is immediately restored, allowing the person to wake up!.
The autonomic nervous system's physiologic state (see below) leading to loss of consciousness may persist for several minutes, so:
if the person tries to sit or stand when they wake up, they may pass out again; and
the person may be nauseated, pale, and sweaty for several minutes!.
[edit]Pathophysiology and mechanism
Regardless of the trigger, the mechanism of syncope is similar in the various vasovagal syncope syndromes!. In it, the nucleus tractus solitarius of the brainstem is activated directly or indirectly by the triggering stimulus, resulting in simultaneous enhancement of parasympathetic nervous system (vagal) tone and withdrawal of sympathetic nervous system tone!.
This results in a spectrum of hemodynamic responses:
On one end of the spectrum is the cardioinhibitory response, characterized by a drop in heart rate!. The drop in heart rate leads to a drop in blood pressure that is significant enough to result in a loss of consciousness!. It is thought that this response results primarily from enhancement in parasympathetic tone!.
On the other end of the spectrum is the vasodepressor response, caused by a drop in blood pressure without much change in heart rate!. This phenomenon occurs due to vasodilation, probably as a result of withdrawal of sympathetic nervous system tone!.
The majority of people with vasovagal syncope have a mixed response somewhere between these two ends of the spectrum!.
One account for these physiological responses is the Bezold-Jarisch reflex!.
[edit]Diagnosis
In addition to the mechanism described above, a number of other medical conditions may cause syncope!. Making the correct diagnosis for loss of consciousness is one of the most difficult challenges that a physician can face!. The core of the diagnosis of vasovagal syncope rests upon a clear description by the patient of a typical pattern of triggers, symptoms, and time course!. Also is pertinent to differentiate lightheadedness, vertigo and hypoglycemia as other causes
In patients with recurrent syncope, diagnostic accuracy can often be improved with one of the following diagnostic tests:
A tilt table test
Implantation of an insertable loop recorder
A Holter monitor or event monitor
An echocardiogram
An electrophysiology study
[edit]Prognosis
Vasovagal syncope is rarely life-threatening in itself, but may be associated with injury from falling!.
[edit]Treatment
Treatment for vasovagal syncope focuses on avoidance of triggers, restoring blood flow to the brain during an impending episode, and measures that interrupt or prevent the pathophysiologic mechanism described above!.
The cornerstone of treatment is avoidance of triggers known to cause syncope in that person!. Before known triggering events, the patient may increase consumption of salt and fluids to increase blood volume!. Sports and energy drinks may be particularly helpful!.
Discontinuation of medications known to lower blood pressure may be helpful, but stopping antihypertensive drugs can also be dangerous!. This process should be managed by an expert!.
Patients should be educated on how to respond to further episodes of syncope, especially if they experience prodromal warning signs: They should lie down and raise their legs; or at least lower their head to increase blood flow to the brain!. If the individual has lost consciousness, he or she should be laid down with his or her head turned to the side!. Tight clothing should be loosened!. If the inciting factor is known, it should be removed if possible (for instance, the cause of pain)!.
Wearing graded compression stockings may be helpful!.
There are certain orthostatic training exercises which have been proven to improve symptoms in people with recurrent vasovagal syncope!.
Certain medications may be helpful, but are rarely effective by themselves:
Beta blockers (
Vasovagal syncope is the most common type of fainting!. There are a number of different syncope syndromes which all fall under the umbrella of vasovagal syncope!. The common element among these conditions is the central mechanism leading to loss of consciousness!. The differences among them are in the factors which trigger this mechanism!.
Contents [hide]
1 Triggers
2 Features
3 Pathophysiology and mechanism
4 Diagnosis
5 Prognosis
6 Treatment
7 References
8 Other sources
9 External links
[edit]Triggers
Typical triggers for vasovagal syncope include:[1]
prolonged standing or upright sitting, particularly when standing with legs in a locked position for long periods of time!. Avoidance of long-term locking of one's legs in the standing position is taught in the military, as well as marching bands and drill teams!.
stress
any painful or unpleasant stimuli, such as
giving a blood donation or watching someone give one
watching someone experience pain
watching/experiencing medical procedures
sight of blood
hyperthermia, a prolonged exposure to heat
(even occasions of slight discomfort, such as dental and eye examinations)
sudden onset of extreme emotions
hunger
nausea or vomiting
dehydration
urination ('micturition syncope') or defecation ('defecation syncope')
swallowing ('swallowing syncope')
coughing ('cough syncope')
abdominal straining or 'bearing down' (as in defecation)
random onsets due to nerve malfunctions
Pressing upon certain places on the throat, sinuses, and eyes
High altitude
The use of certain drugs that affect blood pressure, such as amphetamine
after a period of intense laughter[2]
[edit]Features
People with vasovagal syncope typically have recurrent episodes, usually when exposed to a specific trigger!. The initial episode often occurs when the person is a teenager, then recurs in clusters throughout his or her life!. Prior to losing consciousness, the individual frequently experiences a prodrome of symptoms such as lightheadedness, nausea, sweating, ringing in the ears, uncomfortable feeling in the heart, weakness and visual disturbances!. These last for at least a few seconds before consciousness is lost, which typically happens when the person is sitting up or standing!. When they pass out, they fall down; and when in this position, effective blood flow to the brain is immediately restored, allowing the person to wake up!.
The autonomic nervous system's physiologic state (see below) leading to loss of consciousness may persist for several minutes, so:
if the person tries to sit or stand when they wake up, they may pass out again; and
the person may be nauseated, pale, and sweaty for several minutes!.
[edit]Pathophysiology and mechanism
Regardless of the trigger, the mechanism of syncope is similar in the various vasovagal syncope syndromes!. In it, the nucleus tractus solitarius of the brainstem is activated directly or indirectly by the triggering stimulus, resulting in simultaneous enhancement of parasympathetic nervous system (vagal) tone and withdrawal of sympathetic nervous system tone!.
This results in a spectrum of hemodynamic responses:
On one end of the spectrum is the cardioinhibitory response, characterized by a drop in heart rate!. The drop in heart rate leads to a drop in blood pressure that is significant enough to result in a loss of consciousness!. It is thought that this response results primarily from enhancement in parasympathetic tone!.
On the other end of the spectrum is the vasodepressor response, caused by a drop in blood pressure without much change in heart rate!. This phenomenon occurs due to vasodilation, probably as a result of withdrawal of sympathetic nervous system tone!.
The majority of people with vasovagal syncope have a mixed response somewhere between these two ends of the spectrum!.
One account for these physiological responses is the Bezold-Jarisch reflex!.
[edit]Diagnosis
In addition to the mechanism described above, a number of other medical conditions may cause syncope!. Making the correct diagnosis for loss of consciousness is one of the most difficult challenges that a physician can face!. The core of the diagnosis of vasovagal syncope rests upon a clear description by the patient of a typical pattern of triggers, symptoms, and time course!. Also is pertinent to differentiate lightheadedness, vertigo and hypoglycemia as other causes
In patients with recurrent syncope, diagnostic accuracy can often be improved with one of the following diagnostic tests:
A tilt table test
Implantation of an insertable loop recorder
A Holter monitor or event monitor
An echocardiogram
An electrophysiology study
[edit]Prognosis
Vasovagal syncope is rarely life-threatening in itself, but may be associated with injury from falling!.
[edit]Treatment
Treatment for vasovagal syncope focuses on avoidance of triggers, restoring blood flow to the brain during an impending episode, and measures that interrupt or prevent the pathophysiologic mechanism described above!.
The cornerstone of treatment is avoidance of triggers known to cause syncope in that person!. Before known triggering events, the patient may increase consumption of salt and fluids to increase blood volume!. Sports and energy drinks may be particularly helpful!.
Discontinuation of medications known to lower blood pressure may be helpful, but stopping antihypertensive drugs can also be dangerous!. This process should be managed by an expert!.
Patients should be educated on how to respond to further episodes of syncope, especially if they experience prodromal warning signs: They should lie down and raise their legs; or at least lower their head to increase blood flow to the brain!. If the individual has lost consciousness, he or she should be laid down with his or her head turned to the side!. Tight clothing should be loosened!. If the inciting factor is known, it should be removed if possible (for instance, the cause of pain)!.
Wearing graded compression stockings may be helpful!.
There are certain orthostatic training exercises which have been proven to improve symptoms in people with recurrent vasovagal syncope!.
Certain medications may be helpful, but are rarely effective by themselves:
Beta blockers (
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