Cyst where IV was?!
Question: I had surgery a month ago and when the IV was removed it took forever for the vein to go back to normal. Now there is a little bump under the skin like a bubble close to where the IV needle was inserted. It doesn't really hurt but it's annoying. Could an IV cause a little cyst or bump under the skin. The IV was connected and disconnected many times during my 3 day stay in hospital to allow me to go to the washroom with out a pile of cords. Sometimes the nurses flushed the line other times they didn't. so sometimes I had the joys of small amounts of air being pushed though the line. * grr.
Answers: I had surgery a month ago and when the IV was removed it took forever for the vein to go back to normal. Now there is a little bump under the skin like a bubble close to where the IV needle was inserted. It doesn't really hurt but it's annoying. Could an IV cause a little cyst or bump under the skin. The IV was connected and disconnected many times during my 3 day stay in hospital to allow me to go to the washroom with out a pile of cords. Sometimes the nurses flushed the line other times they didn't. so sometimes I had the joys of small amounts of air being pushed though the line. * grr.
If it is small and not painful, it could just be scar tissue on the wall of the vein.
By the way, the little air bubbles in the tubing will not hurt you at all and they will be reabsorbed by the body.
Since it was a week ago,,it is probably a scar....you vien is healing from the needle prick...one time I was in the hospital for a few week and the iv needle in my hand made my vien collaspe,,,,they had to use the vien in my foot that was very painful...if it dont go away i would get it looked at..sometime you can get an abcess, whick is similar to a cyst (not sure of spelling) and that is much more serious...just keep on eye
Gastrointestinal Imaging
Cyst, choledochal
uncommon congenital disorder characterized by a globular or fusiform dilatation of the common bile duct just below the site of entry of the cystic duct. It is detected with an incidence of 0.2 - 0.5% per 1.000.000 population. There is a 3:1 female predominance. The diameter of the dilatation varies between 2 and several cm. The aetiology is unknown but may be related to an abnormally long common pancreaticobiliary channel. It is assumed that this anomaly could lead to chronic reflux of pancreatic enzymes into the biliary ducts and to recurrent inflammation with resultant stricture formation causing proximal dilatation. Anatomically this disorder is characterized by:
slight dilatation of the extrahepatic biliary ducts proximal to the cystic dilatation due to some degree of obstruction distally;
narrowing of the distal common bile duct; and
abrupt beginning and end of the cystic dilatation.
The wall of the cyst, which is lined by cylindrical or cuboidal epithelium supported by connective tissue and occasional elastic and smooth muscle fibres, varies from 210 mm in thickness. Type I choledocal cyst (Fig.1) refers to aneurysmal dilatation of the common bile duct often accompanied by distal narrowing. It accounts for 80% - 90% of cases.
Types II and III refer to choledocal diverticulum which occurs in 2% of cases and to choledochocoele which involves only the intraduodenal portion of the main bile duct and accounts for 1 - 5% of cases. Choledochocoele is a protrusion of a dilated intramural common bile duct into the duodenum. There are two anatomical types that can not always be differentiated by radiological imaging. In the first, the common bile duct terminates into the choledochocoele which drains into the duodenum via an aperture in its wall. The main pancreatic duct may also empty into the choledochocoele. The second type occurs when the common bile duct enters normally into the papilla and the choledococoele fills and empties directly into the common bile duct. In this variant the pancreatic duct usually enters into the main bile duct proximally to the choledochocoele. Type IV refers to multiple cysts. Type IVa refers to multiple intrahepatic cysts and an extrahepatic cyst and Type IVb refers to multiple extrahepatic cysts. Type V refers to Carolis disease. Classification schemes vary and the definition of Types IV and V are not universally accepted.
Clinical symptoms related to choledochal cysts may appear only at the adult age and are usually caused by complications. Complications of choledochal cysts include: recurrent pancreatitis, cholangitis, biliary stones, intrahepatic abscesses, biliary cirrhosis, portal hypertension and duodenal compression.
Radiological diagnosis
Sonographically the Type I cyst is visible as a fluid containing anechoic structure distinct from the gallbladder but in continuity with the common bile duct. Sonography will also detect associated stones, strictures or carcinoma.
Choledocal cysts are revealed on CT (Fig.2) and MRI as fluid containing, sharply delineated masses in continuity with the extrahepatic bile ducts mostly situated within the pancreatic head. The exact anatomical and topographic situation is better displayed in the coronal than in the transverse plane. This can be accomplished by CT using reformation in the coronal plane with or without the use of cholangiographic contrast medium or by direct coronal imaging with MRI. Optimal and complete anatomical visualization of the anomaly and its topographic relation to the rest of the intra- and extrahepatic bile duct system, particularly in a preoperative setting, requires direct cholangiography (Fig.3).
However, MRCP (Fig.4) nowadays may be able to provide almost identical detailed information as cholangiography without the need of any invasive procedure or of contrast medium. MRCP will also contribute to the differential diagnosis between choledochal cysts and pancreatic cysts or pseudocysts, as well as with enteric duplication cysts. Choledochocoele has different features on cross-sectional imaging than the other types of choledochal cysts. The impression and protrusion of this fluid-filled mass on the medial duodenal wall, as well as its specific intramural localization will suggest the diagnosis (Fig. 5). ERCP optimally displays the anatomical abnormality as well as the repercussion on the bile ducts (Fig. 6).