Myopic degeneration at 30!?!
Question: Myopic degeneration at 30!!?
it is quite rare in someone as young as me!. i was diagnosed recently even though my sight is completely fine at the moment (well fine with glasses) but i wonder if anyone else out there in yahoo land has got this and how soon does deterioration occur!.also, do vitamins containing lutein doe any good!? i am frustrated and upset! Www@Answer-Health@Com
Answers:
Hi there,
Unfortunately, the prognosis for Myopic Degeneration is not particularly good so far as I understand!. I have included some fairly detailed information below!. Are you currently being seen by an Ophthalmologist on a regular basis!? Has any form of treatment been prescribed or offered!?
Degenerative myopia is the seventh leading cause of legal blindness, occurring in about 2% of the U!.S!. population!. It is most often seen in people of Chinese, Japanese, Middle Eastern and Jewish descent!. It is believed to be hereditary!.
Myopic degeneration is similar to AMD (age-related macular degeneration) in that it causes loss of central vision due to degeneration of the photoreceptor cells!. This is caused by separation of the retina as a result of abnormal elongation of the eyeball!. This usually happens because the back of the eye is larger than normal when the eye is very nearsighted!. Marked thinning and stretching may lead to break down of the macula, surrounding retina and it's underlying tissue!. This will cause a varying amount of blurred vision!.
It progresses rapidly, and visual outcome depends largely on the extent of fundus and lenticular changes!. The diagnosis of degenerative myopia is accompanied by characteristic chorioretinal degenerations!. Pathologic myopes, particularly those with higher refractive errors, are at risk for retinal detachment and macular changes!.
People with degenerative myopia have an increased incidence of cataract formation (nuclear cataracts are most typical), Vitreous liquefaction and posterior vitreous detachment, Peripapillary atrophy appearing as temporal choroidal or scleral crescents or rings around the optic disc, Lattice degeneration in the peripheral retina, Tilting or malinsertion of the optic disc, usually associated with myopic conus, Thinning of the retinal pigment epithelium with resulting atrophic appearance of the fundus, Ectasia of the sclera posteriorly (posterior staphyloma), Breaks in Bruch's membrane and choriocapillaris, resulting in lines across the fundus called "lacquer cracks", Fuch's spot in the macular area!.
Even after the eyeball has fully grown (by adulthood), weakness in the sclera (the white outer shell) can lead to development of a posterior staphyloma!.This is a distention of the sclera at the back of the globe where the optic disc and macula are located!. Breaks in Bruch's membrane and atrophy of the choroid layer of the retina (where the blood vessels are) can create lesions known as lacquer cracks!. Blood vessels may protrude through the cracks and leak into the subretinal space beneath the photoreceptor cells!. Known as "choroidal neovascularization," this hemorrhaging can lead to scarring, retinal separation, and profound sight loss in the central field!. If this occurs, immediate treatment is needed, which may be in the form of photodynamic therapy, antiangiogenic drug treatment or both!.
So far as I am aware there are a few areas of research being done at the moment, although I am not up to date on where precisely these treatments have progressed to!.
Kind regardsWww@Answer-Health@Com
Unfortunately, the prognosis for Myopic Degeneration is not particularly good so far as I understand!. I have included some fairly detailed information below!. Are you currently being seen by an Ophthalmologist on a regular basis!? Has any form of treatment been prescribed or offered!?
Degenerative myopia is the seventh leading cause of legal blindness, occurring in about 2% of the U!.S!. population!. It is most often seen in people of Chinese, Japanese, Middle Eastern and Jewish descent!. It is believed to be hereditary!.
Myopic degeneration is similar to AMD (age-related macular degeneration) in that it causes loss of central vision due to degeneration of the photoreceptor cells!. This is caused by separation of the retina as a result of abnormal elongation of the eyeball!. This usually happens because the back of the eye is larger than normal when the eye is very nearsighted!. Marked thinning and stretching may lead to break down of the macula, surrounding retina and it's underlying tissue!. This will cause a varying amount of blurred vision!.
It progresses rapidly, and visual outcome depends largely on the extent of fundus and lenticular changes!. The diagnosis of degenerative myopia is accompanied by characteristic chorioretinal degenerations!. Pathologic myopes, particularly those with higher refractive errors, are at risk for retinal detachment and macular changes!.
People with degenerative myopia have an increased incidence of cataract formation (nuclear cataracts are most typical), Vitreous liquefaction and posterior vitreous detachment, Peripapillary atrophy appearing as temporal choroidal or scleral crescents or rings around the optic disc, Lattice degeneration in the peripheral retina, Tilting or malinsertion of the optic disc, usually associated with myopic conus, Thinning of the retinal pigment epithelium with resulting atrophic appearance of the fundus, Ectasia of the sclera posteriorly (posterior staphyloma), Breaks in Bruch's membrane and choriocapillaris, resulting in lines across the fundus called "lacquer cracks", Fuch's spot in the macular area!.
Even after the eyeball has fully grown (by adulthood), weakness in the sclera (the white outer shell) can lead to development of a posterior staphyloma!.This is a distention of the sclera at the back of the globe where the optic disc and macula are located!. Breaks in Bruch's membrane and atrophy of the choroid layer of the retina (where the blood vessels are) can create lesions known as lacquer cracks!. Blood vessels may protrude through the cracks and leak into the subretinal space beneath the photoreceptor cells!. Known as "choroidal neovascularization," this hemorrhaging can lead to scarring, retinal separation, and profound sight loss in the central field!. If this occurs, immediate treatment is needed, which may be in the form of photodynamic therapy, antiangiogenic drug treatment or both!.
So far as I am aware there are a few areas of research being done at the moment, although I am not up to date on where precisely these treatments have progressed to!.
Kind regardsWww@Answer-Health@Com